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Sildenafil is often a strong and selective inhibitor of cGMP-certain phosphodiesterase style 5 (PDE5), which is chargeable for degradation of cGMP during the corpus cavernosum. The molecular construction of sildenafil is analogous to that of cGMP and acts for a competitive binding agent of PDE5 from the corpus cavernosum, leading to more cGMP and a
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